D. Branch Moody’s laboratory offers NIH-supported Research Fellowships in bacteriology and immunology with a focus on discovery of molecules and genes that promote virulence in pathogenic bacteria and trigger host response.

Successful applicants with an PhD or MD degree will join a collaborative, discovery-focused team in a modern scientific laboratory and receive appointments at the Brigham and Women’s Hospital and Harvard Medical School. We offer high quality training in cellular immunology, bacteriology, chemical biology, and scientific communication. Current projects emphasize whole-organism genetic and metabolic screens to identify virulence factors and the biologically dominant molecules that trigger host macrophage and T cell response. We also offer excellent career development through the Division of Rheumatology, along with scientific programs in the Committee for Immunology at Harvard Medical School.

Existing long-term grants support the full-time salaried positions that pay at or above the NIH scale and includes a competitive benefits package and visa application support.
Interested applicants can learn more about the research by reading recent publications listed below and can apply with a half page statement of interest and CV by writing to Branch Moody .

Recent and representative work:
Discovery of Salmonella phospholipids reveals functional convergence with mycobacteria. JEM, 2019.
M. tuberculosis releases an antacid that remodels phagosomes. Nature Chemical Biology, 2019.
Human γδ T cells recognize CD1b by two distinct mechanisms. PNAS, 2020.
Human skin is colonized by T cells that recognize CD1a independently of lipid. JCI, 2021.
CD1a selectively captures endogenous cellular lipids that broadly block T cell response. JEM, 2021.
Single-cell eQTL models reveal dynamic T cell state dependence of disease loci. Nature, 2022.
Atypical sideways recognition of CD1a by autoreactive γδ T cell receptors. Nature Communications, 2022.
Staphylococcal phosphatidylglycerol antigens activate human T cells via CD1a. Nature Immunology, 2023.
A terpene nucleoside from Mtb induces lysosomal lipid storage in foamy macrophages. JCI, 2023.


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